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I. Stock and B. Wiedemann Table 1. Plesiomonas strains in the present study Origin Strain P-1 CCUG 38281 ; P-2 CCUG 10616D ; P-3 ATCC 14029 ; P-4 CCUG 35490 ; P-5 CCUG 7041A ; P-6 A2000 ; P-7, P-8 P-9 P-10 P-11, P-12, P-13, P-14 P-15 P-16 P-17 P-18, P-19, P-20 P-21, P-22, P-23, P-24, P-25, P-26, P-27, P-28, P-29, P-30, P-31, P-32, P-33, P-34, P-35, P-36, P-37, P-38 P-39 1A ; P-40 2D ; P-41 3A ; P-42 3F ; P-43 4C ; P-44 5A ; P-45 5C ; P-46 5D ; P-47 6A ; P-48 6B ; P-49 7A ; P-50 7B ; P-51 7D ; P-52 8A ; P-53 8C ; P-54 9A ; P-55 10A ; P-56 11A ; P-57 18A ; P-58 19A ; P-59, P-60, P-61, P-62, P-63, P-64, P-65, P-66, P-67, P-68 P-69 LMG 4243 ATCC 14030 ; P-70 LMG 4244 ; P-71 LMG 4245 ; P-72 LMG 4246 ; Serovar unknown unknown unknown unknown unknown unknown unknown unknown unknown unknown O2: H1a, 1c O5: H4 O11: H2 unknown unknown material human purulent tendovaginitis aquarium, sludge dog canine appendicitis human faeces human faeces, fulminant diarrhoea human clinical specimen human faeces human clinical specimen human faeces human clinical specimen human clinical specimen human clinical specimen human clinical specimen human clinical specimen country Sweden Sweden USA Sweden Sri Lanka Germany France Belgium Switzerland UK USA USA USA Germany USA Source E. Valsen, Swedena E. Valsen, Swedena E. Valsen, Swedena E. Valsen, Swedena E. Valsen, Swedena H. Schaal, Germanyb A. v. Graevenitz, Switzerlandc A. v. Graevenitz, Switzerlandc A. v. Graevenitz, Switzerlandc A. v. Graevenitz, Switzerlandc A. v. Graevenitz, Switzerlandc A. v. Graevenitz, Switzerlandc A. v. Graevenitz, Switzerlandc A. v. Graevenitz, Switzerlandc A. v. Graevenitz, Switzerlandc.
Associated with a fracture of the mandible. J Oral Surg 27: 145-149, Feb 1969 Hagadorn B, Smith HW, Rosnagle RS: Cervical spine osteomyelltis secondary to a foreign body in the hypopharynx. Arch.
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Is the equivalent of a 5th level spell. Yellow Veil: An initiate of 3rd level or higher can create a yellow veil. This veil prevents gasses or clouds from entering the warded area, and it defeats petrification attacks. In addition, a character inside a personal or area warding imbued with a yellow veil has immunity to poison introduced from outside the warding. A creature crossing a yellow veil takes 80 points of electricity damage Reflex half ; . A disintegrate spell destroys the yellow veil but is negated in the process. A warding with this veil is equivalent to a 6th level spell. Green Veil: At 4th level and higher, an initiate masters the green veil. This veil stops the passage of breath weapons. A creature crossing the green veil must succeed on a Fortitude save or die; on a successful save, the creature takes 1d6 points of Constitution damage. This veil is a poison effect. A passwall spell destroys a green veil. A warding with this veil is considered a 6th level spell. Blue Veil: At 5th level, an initiate learns the blue veil. The veil blocks all divinations and mind affecting spells and abilities. Any creature crossing a blue veil must succeed on a Fortitude save or be petrified. A magic missile spell destroys a blue veil but it is negated by it. A warding with this veil is considered a 6th level spell. Indigo Veil: A 6th level initiate can create the mighty indigo veil. This veil prevents the passage of spells and abilities. Any creature crossing this veil must succeed on a Will save or become confused, as if by an insanity spell. A daylight spell negates and is negated by the indigo veil. A warding with this veil is considered a 7th level spell. Violet Veil: At 7th level, an initiate masters the seventh and final veil: the violet veil. This barrier destroys all objects and effects that cross it, as if they were disintegrated. Living creatures passing a violet veil must succeed on a Will save or be shifted to a random place on a random plane. A violet veil is destroyed by a successful dispel magic spell. A warding of this veil is the equivalent of an 8th level spell. Unimpeachable Abjuration Ex ; : An initiate's abjuration spells are particularly difficult to defeat with spells or effects that dispel them. An initiate can add her class level to the DC to dispel any abjuration spell or effect she creates. Unanswerable Strike Ex ; : Due to her study of magical defenses, an initiate learns how to defeat them more easily. At 2nd level and higher, she gains a + 2 bonus on caster level checks to counter or dispel abjuration spells. At 6th level, this bonus increases to a + bonus. Reactive Warding Sp ; : At 4th level, an initiate learns to create a warding in response to an attack. She can raise a warding as an immediate action, after an opponent begins an action but before it is completed. For example, if she sees an enemy warrior charging her, she can raise a warding to protect herself. The opponent can choose to continue the charge through the warding or can halt outside. Double Warding: At 6th level and higher, an initiate can raise two veils at once any time she creates a warding. This still counts as only one use of her warding ability. The less powerful effect progressing from red up through violet ; is always considered to be outside the more powerful effect, so a double warding consisting of a blue veil and a green veil would subject any creature passing through to the green veil first, followed by the blue veil. To negate the entire warding, the outermost veil must be negated before the innermost can be negated. Kaleidoscopic Doom Sp ; : At 7th level, an Initiate of the Sevenfold Veil learns the secret of the awesome kaleidoscopic doom. Once per day as a standard action, she designates one creature within 60 feet and turns magical effects currently affecting the creature against it. This effect functions like a targeted greater dispel magic, except that for every spell or effect negated on the target, the effect of one veil is visited on the victim as if the subject had crossed it. The veils created around the victim proceed through the spectrum from red to violet, with one veil activated per spell negated. Thus a creature with three spells negated would be subject to the effects of the red, orange and yellow veils. The subject is still entitled to the normal saving throws allowed by each veil. This ability is the equivalent of a 9th level spell.
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Using the segmentation of the two 2D color planes, as described in the previous subsection, one of the 11 color categories is assigned to every point i.e., color ; in the whole 3D color space. The validation of this categorization method consisted of two tests and the analysis of their results: i ; categorization of non-fuzzy colors and ii ; categorization of fuzzy colors. The segmented color space is considered valid if and only if it categorizes the stimuli used in the experiments to the same categories as the subjects did. The non-fuzzy colors are those colors that are categorized consistently to one color category by the participants of the experiments, as described in Chapter 3. The fuzzy colors are those colors categorized to two or more ; categories by at least 10 subjects. The fuzzy and non-fuzzy colors together make up the set of CLUT markers, derived from the experiments. In Table 5.1, the 11 color categories are listed. The segmented color space has a 100% match with the experimental results. All non-fuzzy colors are categorized correctly. All fuzzy colors are mapped in one of the categories to which they were assigned to in the experiments. In Figure 5.5a, the non-fuzzy CLUT markers and the calculated borders of the chromatic categories have been plotted. In Figure 5.5b, the fuzzy CLUT markers in the plane used for chromatic segmentation, have been visualized. Since the hue and intensity axis are able to describe non-overlapping clusters for chromatic categories, this 2D approximation was appropriate to segment the color space with FEED using only hue and intensity values ; . Figure 5.5b shows that fuzzy data points are close to the calculated borders. Please note that in both Figure 5.5a and in Figure 5.5b some data points 80 and colesevelam
Results suggested that CD4 + CD25high T cells were not per se incapable to proliferate, we next examined whether their long term and large scale expansion could be achieved under adequate co-stimulatory conditions. We applied a previously described culture system in which stimulatory antibodies are presented by Fc RII CD32 ; -bearing L cells.32 Neither CD4 + CD25high nor CD4 + CD25 T cells proliferated upon co-culture with L cells alone or additional IL-2. However, the combined stimulation via CD3 and CD28 in the presence of CD32 + L cells was sufficient to induce proliferation in both populations, which was further enhanced by IL-2 and reached maximum levels at 100 1000 U ml Fig. 2A and data not shown ; . In consequence, saturating doses of 300 U ml IL-2 were used in all subsequent experiments. CD4 + CD25high T cells cultured under these conditions expanded dramatically with an average 13, 000-fold range: 1, 250 - 39, 600-fold ; increase in cell numbers within 3-4 wks n 9 cultures from separate leukaphereses of 5 donors ; Fig. 2B ; . Although CD4 + CD25high T cell expansion usually slowed down after 4 wks, individual cultures were propagated for more than 3 months with a maximum expansion rate of 1.2 109. Expansion of CD4 + CD25 T cells from the same donors cultured in parallel usually exceeded that of CD4 + CD25high T cells and resulted in an average 28, 900-fold expansion range 2, 200 - 56, 900-fold ; within 3-4 wks n 9 cultures from separate leukaphereses of 5 donors, data not shown ; . To differentiate whether CD4 + CD25high T cell expansion was polyclonal or driven by individual T cell clones, TCR V -analysis was performed. CD4 + CD25high T cells within unmanipulated PBMC showed a polyclonal V -usage similar to that of CD4 + CD25 T cells Fig. 2C ; . After isolation and in vitro expansion for 25 d, their TCR V -repertoire neither differed significantly from that of freshly isolated CD4 + CD25high T cells nor from that of freshly isolated or expanded CD4 + CD25 T.
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Results- No adverse of treatment were observed. Conclusions These results suggest a significant role for octreotide as an adjunct to intravenous dextrose in the management of severe and refractory cases of sulfonylurea-induced hypoglycaemia. EXERCISE Effect of thermal biofeedback-assisted relaxation training on blood circulation in the lower extremities of a population with diabetes Rice B.I. and Schindler J.V. Diabetes Care 1992; 15: 853-8. Objective- Investigate the effect of relaxation training thermal biofeedback on blood circulation in the lower extremities of diabetic subjects. Research Design and Methods- Diabetic subjects n 40 ; aged 17-73 yr were volunteers recruited through the University of Wisconsin-La Crosses, the local ADA Chapter, and a medical clinic. A withinsubjects experimental design was used. During phase 1 , all subjects used a self-selected relaxation method and recorded toe temperatures daily. During phase 2, subjects were taught a biofeedback-assisted relaxation technique designed to elicit sensations of warmth in the lower extremities and increase circulation and temperature. Subjects relaxed at home with the use of designed relaxation tape. They measured and recorded toe temperatures. Each phase lasted 4 wk. Results- Toe temperature and blood volume pulse BVP ; data were gathered at the beginning and end of phases 1 and 2 . Paired t tests compared the means of temperature per cent change scores between 1 and 2. Mean temperature change score were 8.73% phase 1 ; and 31.38% phase 2 ; t 8.00, df 39 , P 0.001 ; . Mean BVP change scores were 2.33% phase 1 ; and 22.47% phase 2 ; t 9.24, df 35, P 0.001 ; . Based on eta square, 71% of the BVP increase in phase 2 was attributed to the and colestipol.
We met as the Canadian Public Health Laboratory Forum CPHLF ; for the first time in 2001. I grateful to Dr. Greg Horsman, Saskatchewan Public Health Laboratory and Drs. Frank Plummer and Amin Kabani, National Microbiology Laboratory, for their support and work on this strategic partnership. CPHLF partners with the public health laboratory Directors in the US on International issues.
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To compare young versus aged rats for differences in the percentage of small infarcts displaying hypercellularity or hypocellularity, a ratio estimate approach from sampling theory27 was used. For each group the number of infarcts varies. It would be inappropriate to assume that the hypercellularity status of infarcts was independent across all animals. Hence, we used a technique that adjusts for the dependence within animals.28 This approach assumes that each animal is a cluster and computes estimates of the percentages and variances across clusters. For testing the two groups, an estimate of the covariance was also computed. This approach was also used to analyze the relative size of infarcts for different comparison groups of interest. All tests reported are two-sided, and ? 0.05 was considered significant except where noted. Results All six of the aged Fisher rats irradiated for 20 minutes either died or were killed because they appeared near death from 2 to 48 hours after irradiation. Three of the 11 aged rats irradiated for 15 minutes were eliminated from the series; one had adhesive arachnoiditis, and two died before the scheduled sacrifice time within 8 hours ; . The remaining eight aged rats irradiated for 15 minutes survived to the protocol sacrifice time. All 16 young rats, 12 irradiated for 20 minutes and four irradiated for 15 minutes, survived 4 days. There were 142 cerebral infarcts in the 24 rats, 68 infarcts in eight aged rats and 74 infarcts in 16 young rats. The numbers, sizes, and distributions of these lesions are listed in Table 1. The mean number of infarcts was 5.9 per rat, with a range of 0-16 per rat. The distribution of infarct number was not normal, with eight of 24 rats having 0-2 infarcts and four of 24 rats having 14-16 infarcts. Infarcts 1 mm average dimension ; in size in young rats were more often hypercellular compared with surrounding normal tissue Figure 1A ; . Astro.
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Introduction Several aspects of insect feeding behaviour have been studied intensively. Although present knowledge of the physiology of insect feeding does not allow a detailed description of this type of behaviour in its entirety, attempts have been made to understand food intake as a function of a large set of physiological and external variables such as the size and nutritional quality of the previous meal, endogenous rhythms, food stimuli and the light regime for a review, see Simpson, 1990 ; . According to these sources, feeding behaviour does not occur randomly in locusts but consists of groups of `meals' that may include short intra-meal gaps. The phases of food intake within each meal are characterized by regular rhythmical action of the mandibular closer muscle. Despite these studies, little information is available on the neuroethological basis of feeding behaviour, especially on the components of the neural pattern generators controlling the mouthparts. In insects, such investigations have been impeded by the inaccessibility of the suboesophageal ganglion. The massive dissection required to gain access to the ganglion usually abolishes all neural activity related to feeding. This problem was partially solved in larvae of the tobacco hornworm Manduca sexta, where cutting the neck connectives disinhibits the chewing motor progamme and thus allows electrophysiological studies in isolated suboesophageal ganglia Rowell and Simpson, 1992 ; . On the basis of this work and that of Griss et al. 1991 ; , some mandibular motoneurones and commit
INTRODUCTION Phenolic compounds are important components of many fruits, vegetables, and beverages contributing to their colour and sensory properties. Epidemiological studies have demonstrated that the composition of phenol-rich food retards the progression of arteriosclerosis and reduces the incidence of heart diseases by preventing the oxidative stress, that is, lipid peroxidation in arterial macrophages and in lipoproteins [1, 2]. More recently, some authors reported that anthocyanins decreased cadmium accumulation in liver and kidney, the concentration of bilirubin and urea in blood serum, and aspartate aminotransferase and alanine aminotransferase activities [3]. Pomegranate juice is an important source of phenolic compounds, with anthocyanins being one of the most important, especially the 3-glucosides and 3, 5diglucosides of delphinidin, cyanidin, and pelargonidin [4]. These components along with gallagyl-type tannins, ellagic acid derivatives, and other hydrolysable tannins could contribute in some way to the antioxidant activity of pomegranate juice [2]. "Assaria" pomegranate is a Portuguese variety cultivated in the southern region of the country. Its edible seeds are a favourite snack due to sweet taste and tenderness, and its fruits are mainly used for direct consump
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Untoward changes due to the greater stress of cerebral ischemia induced by bilateral ligation. Many animals succumbed soon after the second carotid artery was ligated. However, as the animals began to recover from the anesthesia they promptly showed severe blanching of both eyes, many manifested violent convulsions and died, many became paraplegic, and practically all showed marked extensor rigidity. As in our previous report, 1 temporary fatty steatosis of the liver developed in all the animals fig. 1 ; . Twenty-one percent of the arteriosclerotic male breeder rats exhibited grossly visible myocardial infarction; none of these arteriosclerotic males showed any gross evidence of their arterial disease. At autopsy, 8 3 % of the female breeders displayed grossly visible arteriosclerosis ranging from minimal to and copaxone.
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