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References 1 Shee JC. Dangerous potentiation of pethidine by iproniazid, and its treatment. BMJ 1960; 2: 507-9. Vigran IM. Dangerous potentiation of meperidine hydrochloride by pargyline hydrochloride. JAMA 1964; 187: 953-4. Stack CG, Rogers P, Linter SPK. Monoamine oxidase inhibitors and anaesthesia. A review. Br J Anaesth 1988; 60: 222-7. McFarlane HJ. Anaesthesia and the new generation monoamine oxidase inhibitors. Anaesthesia 1994; 49: 597-9. Rivers N, Homer B. Possible lethal reaction between Nardil and dextromethorphan Letter ; . Can Med Assoc J 1970; 103: 85. Powell H. Use of alfentanil in a patient receiving monoamine oxidase inhibitor therapy Letter ; . Br J Anaesth 1990; 64: 528. Churchill-Davidson HC. Anaesthesia and monoamine oxidase inhibitors Letter ; . BMJ 1965; 1: 520. James FM III, Greiss FC Jr, Kemp RA. An evaluation of vasopressor therapy for maternal hypotension during spinal anesthesia. Anesthesiology 1970; 33: 25-34. Tong C, Eisenach JC. The vascular mechanism of ephedrine's beneficial effect on uterine perfusion during pregnancy. Anesthesiology 1992; 76: 792-8. Stockley IH. Drug Interactions: A Source Book of Adverse Interactions, Their Mechanisms, Clinical Importance and Management. Oxford: Blackwell Scientific Publications, 1981. 11 Boakes AJ, Laurence DR, Teoh PC, Barar FSK, Benedikter LT, Prichard BNC. Interactions between sympathomimetic amines and antidepressant agents in man. BMJ 1973; 1: 311-5.
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NMHC Maintenance Drug List for Sound Health & Wellness Trust Created 01 08 2008 This list includes those drugs and products that Medispan designates as maintenance, as well as those products that Sound Health specifies as maintenance drugs. Thus, this is a general list and must be interpreted in terms of specific Sound Health & Wellness Trust coverage. Tier 3 are those drugs that will have two copays for 60 to 90 days at the mail at retail program. Restricted distribution drugs are only dispensed at designated specialty pharmacies not in the network unless indicated. Product Name CODITUSS DM COLD & ALLERGY COLD & COUGH DM COLD COUGH DM CHILDRENS COLDEC DM COLDEC-DM COLFED-A COPHENE #2 CORFEN-DM CORYZA-DM CP DEC-DM CVS CHILDREN'S MULTI-SYMP CVS COLD COUGH DM CHILDRE D-TANN CT D-TANN DM DALLERGY DM DE-CHLOR DM DE-CHLOR DR DEC-CHLORPHEN DM DECON DM DECONAMINE DECONAMINE SR DECONGESTANT + DECONGESTANT PLUS DECONSAL DM DELTUSS DMX DEX PC DEXTROMETHORPHAN CHLORPHE DEXTROMETHORPHAN PHENYLEP DI BROMM COLD ALLERGY DICEL DM DIHISTINE DH DIHISTINE DH DIMAPHEN DIMAPHEN DM DIMAPHEN DM COLD COUGH CH DIMETAPP DIMETAPP DM COLD & COUGH DM TANN PE TANN BROM TANN DM-PE-CHLOR DONATUSSIN DM DUR-TANN DM DUR-TANN FORTE DURAGANIDIN NR DURATAN DM DURATAN FORTE DURATUSS AC 12 DURATUSS DA DYNAHIST ER DYTAN-CD DYTAN-CS DYTAN-DM ED DM ED-A-HIST DM ENDACOF-DM Therapy Class COUGH COLD ALLERGY COUGH COLD ALLERGY COUGH COLD ALLERGY COUGH COLD ALLERGY COUGH COLD ALLERGY COUGH COLD ALLERGY COUGH COLD ALLERGY COUGH COLD ALLERGY COUGH COLD ALLERGY COUGH COLD ALLERGY COUGH COLD ALLERGY COUGH COLD ALLERGY COUGH COLD ALLERGY COUGH COLD ALLERGY COUGH COLD ALLERGY COUGH COLD ALLERGY COUGH COLD ALLERGY COUGH COLD ALLERGY COUGH COLD ALLERGY COUGH COLD ALLERGY COUGH COLD ALLERGY COUGH COLD ALLERGY COUGH COLD ALLERGY COUGH COLD ALLERGY COUGH COLD ALLERGY COUGH COLD ALLERGY COUGH COLD ALLERGY COUGH COLD ALLERGY COUGH COLD ALLERGY COUGH COLD ALLERGY COUGH COLD ALLERGY COUGH COLD ALLERGY COUGH COLD ALLERGY COUGH COLD ALLERGY COUGH COLD ALLERGY COUGH COLD ALLERGY COUGH COLD ALLERGY COUGH COLD ALLERGY COUGH COLD ALLERGY COUGH COLD ALLERGY COUGH COLD ALLERGY COUGH COLD ALLERGY COUGH COLD ALLERGY COUGH COLD ALLERGY COUGH COLD ALLERGY COUGH COLD ALLERGY COUGH COLD ALLERGY COUGH COLD ALLERGY COUGH COLD ALLERGY COUGH COLD ALLERGY COUGH COLD ALLERGY COUGH COLD ALLERGY COUGH COLD ALLERGY COUGH COLD ALLERGY COUGH COLD ALLERGY Rx OTC Tier 3 Restricted Distribution OTC OTC OTC OTC RX RX RX OTC OTC RX RX RX OTC OTC RX RX RX OTC RX OTC RX OTC OTC OTC OTC OTC RX RX RX.
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Updated information and services can be found at: : bloodjournal.hematologylibrary cgi content full 107 3 924 Articles on similar topics may be found in the following Blood collections: Stem Cells in Hematology 165 articles ; Hematopoiesis 2337 articles ; Information about reproducing this article in parts or in its entirety may be found online at: : bloodjournal.hematologylibrary misc rights.dtl#repub requests Information about ordering reprints may be found online at: : bloodjournal.hematologylibrary misc rights.dtl#reprints Information about subscriptions and ASH membership may be found online at: : bloodjournal.hematologylibrary subscriptions index.dtl and diamox.
16 that the underlying theoretical basis for such indices should be established and understood in order to allow for the contaminating effects of other pharmacokinetic variables. In particular, the impact of divergent primary metabolic pathways mediated by different enzymes but leading to the formation of the same secondary metabolite via the same enzymes should be considered. For example, the ratios of 5-hydroxyomeprazole to omeprazole and of omeprazole sulphone to omeprazole have been proposed as convenient markers of CYP2C19 and 3A4 activities, respectively. However, both of the products are further metabolised to a common secondary metabolite by the opposite enzyme. Accordingly, any change of either ratio may reflect induction of one pathway and or inhibition of the other. Also, it should be recognised that metabolic ratios depend upon renal clearance, of the parent drug in the case of urinary ratios and of the metabolite in the case of plasma ratios. If drug or metabolite are lipid-soluble as in the case of dextromethorphan and metoprolol ; , diurnal variation of urinary pH can make a significant contribution to the intra-subject variability of these ratios. Clearly, this has implications for the choice of probe substrate and the sensitivity of the associated metric to pick up a drug interaction. The `best buy' with respect to indirect metrics for some enzymes has been established. For example, the 5 7 h plasma or saliva paraxanthine caffeine ratio has been established theoretically and by experiment to be the most robust indirect index of CYP1A2 activity. A systematic examination of the robustness of all indirect metrics is needed in order to guide standardisation. Should we insist on conventional `goalposts' when using the confidence interval approach? It was generally agreed that the outcome of a drug drug interaction study should be evaluated statistically in the same way as bioequivalence studies, i.e. based on the confidence intervals of differences. While it was considered reasonable as a default position for most drugs to conclude `no interaction' if the 90% confidence interval of the ratio of log AUC or Cmax in the absence and presence of inhibitor is within the conventional `goalposts' 0.80 1.25 for AUC; 0.70 1.43 for Cmax ; , it was emphasised that the limits should be flexible depending upon pharmacodynamic and clinical considerations. However, as with bioequivalence, establishing what the exact limits should be for specific drugs is not easy. At least a doubling of systemic exposure in the presence of an inhibitor was agreed be a reasonable basis for labelling action. Does population pharmacokinetics help? The application of population kinetics to detect drug drug interactions was considered to be useful and complementary to in vitro studies and small n studies in healthy subjects, providing due care is given to the issue of statistical power. Non-positive findings should be interpreted appropriately as indicating failure to detect an interaction rather than the lack of such.
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Measured using the first menstrual cycle for the extended regimen group and the third menstrual cycle for the cyclic regimen group, was significantly longer in the extended regimen group than in the cyclic regimen group 6.9 compared with 5.2 days, P .001 ; . Overall satisfaction, evaluated on day 84, was similar between groups; 86.3% 107 124 ; of subjects in the extended regimen group and 88.6% 62 70 ; of subjects in the cyclic group were very or somewhat satisfied. Subjects in the extended regimen group indicated a perception of lighter or no bleeding flow compared with their pretreatment menstrual flow. On day 84, 35% 43 ; of the subjects in the extended regimen group reported that they had not bled, compared with 3% 2 70 ; of cyclic subjects. For the overall global assessment of the study drug regimen, ratings of "excellent" or "good" were given by 87% 70 80 ; and 79% 122 155 ; of subjects in the cyclic and extended treatment regimens, respectively.
Throughout development, whereas in human PAP, GM-CSF is present but is neutralized by circulating levels of anti-GM-CSF which appears most commonly in the second or third decade of life 12 ; . We have shown here that BAL cells from PAP patients and diflunisal.
The use of bisphosphonates have been well established in the treatment of osteoporosis.8, 22 Alendronate and risedronate are approved for the prevention and treatment of osteoporosis not due to cancer treatment; 21, 61 and have been shown to decrease bone resorption and increase bone mass.
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Alto, Calif: Consulting Psychologists Press Inc, 1970 14 Kirsch JL, Muro JR, Stansbury DW, Fischer CE, Monfore R, Light RW. Effect of naloxone on maximal exercise performance and control of ventilation in COPD. Chest 1989; 96: 761-66 Giron AE, Stansbury DW, Fischer CE, Light RW. Lack of effect of dextromethorphan on breathlessness and exercise performance in patients with chronic obstructive pulmonary disease COPD ; . Eur Respir Dis J 1991; 4: 532-35 Borg GAY Psychophysical basis of perceived exertion. Med Sci Sports Exer 1982; 14: 377-81 McCavin CR, Gupta SP, McHardy GJR. Twelve-minute walking test for assessing disability in chronic bronchitis. Br Med J 1976; 1: 822-23 McCavin CR, Artvinli M, Naoe H, McHardy GJ. Dyspnoea, disability, and distance walked: comparison of estimates of exercise performance in respiratory disease. Br Med J 1978; 2241-43 19 Beck AT, Ward CH, Mendelson M, Mock J, Erbaugh J. An inventory for measuring depression. Arch Gen Psychiatry 1961; 4: 561-71 Blackie SP, Fairbarn MF, McElvaney GN, Monison NJ, Wilcox PG, Pardy RL. Prediction of maximal oxygen uptake and power during cycle ergometry in subjects older than 55 years of age. Rev Respir Dis 1989; 139: 1424-29 Bohm C, Robinson DS, Gammans RE, Shrotriya RC, Alms DR
Geriatric Use: Ages 65 and older ; Geriatric patients taking sympathomimetics may be more likely to experience confusion, hallucinations, seizures, and central nervous system CNS depression. Geriatric patients may also be more sensitive to the effects, especially to the vasopressor effects, of sympathomimetic amines. Demonstrate safe use of a short-acting sympathomimetic formulation before use of a sustained-action formulation in elderly patients. ADVERSE REACTIONS: Pseudoephedrine may cause mild central nervous system stimulation, especially in those patients who are hypersensitive to sympathomimetic drugs. Nervousness, excitability, restlessness, dizziness, weakness and insomnia may also occur. Headache and drowsiness have also been reported. Large doses may cause lightheadedness, nausea and or vomiting. Sympathomimetics have been associated with certain untoward reactions including fear, anxiety, nervousness, restlessness, tremor, weakness, pallor, respiratory difficulty, dysuria, insomnia, hallucinations, convulsions, CNS depression, arrhythmias, and cardiovascular collapse with hypotension. Adverse effects associated with dextromethorphan are generally infrequent and mild. Products containing dextromethorphan have been associated with nausea, dizziness, fatigue, gastrointestinal disturbances, and skin eruptions. Chlorpheniramine may cause slight to moderate drowsiness and is the most frequent side effect. Other possible side effects of antihistamines include: General: Urticaria, drug rash, anaphylactic shock, photosensitivity, excessive perspiration, chills, dryness of mouth, nose and throat; Cardiovascular: Hypotension, headache, palpitation, tachycardia, extra systoles; Hematological: Hemolytic anemia, thrombocytopenia, agranulocytosis; CNS: Sedation, dizziness, disturbed coordination, fatigue, confusion, restlessness, excitation, nervousness, tremor, irritability, insomnia, euphoria, paresthesia, blurred vision, diplopia, vertigo, tinnitus, hysteria, neuritis, convulsion; Gastrointestinal: Epigastric distress, anorexia, nausea, vomiting, diarrhea, constipation; Genitourinary: Urinary frequency, difficult urination, urinary retention, early menses; Respiratory: Thickening of bronchial secretions, tightness of chest, wheezing and nasal stuffiness. DRUG ABUSE AND DEPENDENCE: Pseudoephedrine, like other CNS stimulants, has been abused. At high doses, subjects commonly experience an elevation of mood, a sense of increased energy and alertness, and decreased appetite. Some individuals become anxious, irritable, and loquacious. In addition to the marked euphoria, the user experiences a sense of markedly enhanced physical strength and mental capacity. With continued use, tolerance develops, the user increases the dose and toxic signs and symptoms appear. Depression may follow rapid withdrawal. Narcotic antitussives and stimulants, such as pseudoephedrine, are banned and tested for by the U.S. Olympic Committee USOC ; and the National Collegiate Athletic Association NCAA and dilaudid.
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Postfire years. Observed postfire erosion rates depend on the particular sequence of fire and winter rains. For example, in the Bell IV drainage basin on the San Dimas Experimental 3 Forest, about 108 m ha of sediment were discharged in the first 2 postfire years fig. 3 ; . The first year was very dry and the second had normal rainfall. In contrast, nearby Harrow 3 Canyon--which burned in 1968--produced 438 m ha of sediment as the result of a storm having a.
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Pretreatment evaluation included a complete history and clinical examination, full blood count, biochemical profile, urinalysis and electrocardiogram. In addition, a chest X-ray and or computed tomography scan of the abdomen and other sites of disease was performed before starting chemotherapy. During therapy, patients had a weekly toxicity assessment, full blood count and biochemical profile. Physical examination and World Health Organization WHO ; performance status were recorded before each cycle or more frequently if clinically indicated ; . Tumour assessments were performed after three and six cycles of therapy . Response was determined according to WHO criteria [17]. All responses were to be documented with a repeat evaluation after 4 weeks and dextromethorphan.
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