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10. Nakagawa M, Stites DP, Farhat S et al. T cell proliferative response to human papilloma virus type 16 peptides: relationship to cervical intra-epithelial neoplasia. Clin Diagn Lab Immunol 1996; 3: 205 Takeshita S, Breen EC, Ivashchenko M et al. Induction of IL-6 and IL-10 production by recombinant HIV-1 envelope glycoprotein 41 gp41 ; and the THP-1 human monocytic cell line. Cell Immunol 1995; 165: 234242. Maiman M, Fruchter RG, Serur E, Boyce JG. Prevalence of human immunodeficiency virus in a colposcopy clinic. JAMA 1988; 260: 22142215. Holcomb K, Maiman M, Dimaio T, Gates J. Rapid progression to invasive cervix cancer in a woman infected with the human immunodeficiency virus. Obstet Gynecol 1998; 91: 848850. Belafsky P, Clark RA, Kissinger P, Torres J. Natural history of low-grade squamous intra-epithelial lesions in women infected with human immunodeficiency virus. J Acquir Immune Defic Syndr Hum Retrovirol 1996; 11: 511512. Robinson W 3rd. Invasive and preinvasive cervical neoplasia in human immunodeficiency virus-infected women. Semin Oncol 2000; 27: 463470. Palefsky JM. Anal squamous intra-epithelial lesions in human immunodeficiency virus-positive men and women. Semin Oncol 2000; 27: 471479. Ateenyi-Agaba C. Conjunctival squamous-cell carcinoma associated with HIV infection in Kampala, Uganda. Lancet 1995; 345: 695696. Goedert JJ, Cote TR. Conjunctival malignant disease with AIDS in USA. Lancet 1995; 346: 257258. Frisch M, Biggar RJ, Goedert JJ. Human papillomavirus-associated cancers in patients with human immunodeficiency virus infection and acquired immunodeficiency syndrome. J Natl Cancer Inst 2000; 92: 15001510. Sanders CJ. Condylomata acuminata of the penis progressing rapidly to invasive squamous cell carcinoma. Genitourin Med 1997; 73: 402 Yoganathan K, Patel RN, Maitland N et al. Carcinoma of the penis in an HIV positive patient. Genitourin Med 1995; 71: 4142. Bernardi D, Salvioni R, Vaccher E et al. Testicular germ cell tumors and human immunodeficiency virus infection: a report of 26 cases. Italian Cooperative Group on AIDS and Tumors. J Clin Oncol 1995; 13: 27052711. Ratner L, Redden D, Hamzeh F et al. Chemotherapy for AIDS associated non-Hodgkin's lymphoma in combination with highly active retroviral therapy HAART ; is not associated with excessive toxicity. Third National AIDS Malignancy Conference, Bethesda, MD, 1999. J AIDS 1999; 21: A32.
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Choriocarcinoma n 3 ; . The median age was 28 years old range: 2340 ; and all patients were under the same oral contraception with 50 g of ethinyl oestradiol ; . Diagnoses were reconfirmed by review of histological slides. This study was approved by the local ethics committee. Chemotherapy consisted of a combination of dactinomycin and etoposide every week for the treatment of hydatidiform and invasive mole, and a combination of dactinomycin and etoposide every 2 weeks plus cisplatin every 4 weeks for choriocarcinoma and for methotrexate-resistant hydatidiform mole. Serum inhibins A and B were measured using a two-site sandwich ; enzyme-linked immunoassay ELISA; Serotec, Oxford, UK ; . These tests are based on specific monoclonal antibodies raised against the , A and B subunits. Briefly, each assay was performed as follows: serum samples were first mixed with a 10% sodium dodecyl sulphate solution and incubated at 100C for 3 min. After cooling, a hydrogen peroxide solution was added to tubes for a further incubation at room temperature. Samples were then transferred to antibody-coated microtitre plates and incubated at room temperature for 2 h for inhibin A ; and overnight for inhibin B ; , according to the manufacturer's instructions. Recombinant inhibin A and inhibin B were used as standards. The inhibin A and B detection limits were 4 and 10 ng l respectively. Serum HCG and free HCG were measured using specific immunoradiometric assays Cisbio International, Giff sur Yvette, France ; Ozturk et al., 1987 ; . The intra- and interassay coefficients of variation were 4% and 7% for inhibin A, and 4% and 8% for inhibin B respectively.
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Symptom Text: 8 days after anthrax and smallpox vaccines, developed vesicular rash on trunk, neck, scalp and bilateral upper extremities. Patient was afibrile and had no other symptoms. Was see in Emergency Room and diagnosed as generalized vaccinia. He was hospitalized because he lived in an open-bay barracks and was felt to be infectious. Follow up from Hospital Discharge: headache, nausea, malaise. None Other Meds: Lab Data: History: Prex Illness: Prex Vax Illns: None None.
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Clinical trials for malignant lymphoma vorable factors affecting PFS all P 0.05 ; . The PFS intervals of patients with higher serum rituximab levels 70 g ml ; immediately before the third infusion were longer than other patients P 0.05 ; . In summary, rituximab is a highly effective agent in relapsed indolent B-NHL and MCL with acceptable toxicities. Rituximab is more effective in the treatment of relapsed indolent BNHL than of MCL, and in patients without extranodal disease or with a history of having received only one prior chemotherapy regimen. Several prognostic factors and serum rituximab levels are useful in predicting the efficacy of rituximab monotherapy 16 ; . SUBSEQUENT CLINICAL TRIALS OF RITUXIMAB IN JAPAN In 1999, two multicenter phase II studies were initiated in Japan: a single-agent phase II study in recurrent aggressive BNHL 18 ; , and a randomized phase II study of rituximab in combination with CHOP, comparing concurrent and sequential administration in previously untreated advanced-stage indolent B-NHL 43 ; . In the single-agent phase II study of rituximab for recurrent aggressive B-NHL, a total of 68 patients with REAL types II3, 9, 10 and 11 were enrolled and treated with rituximab 375 mg m2, weekly for 8 consecutive weeks 18 ; . A central pathology review disclosed that 57 patients 84% ; were eligible. The Independent Computed Tomography Review Committee confirmed that ORR was 35% 24 68 ; in enrolled patients and 37% 21 57 ; in eligible patients. The median PFS of the 53 evaluable patients was 52 days, whereas the time to progression of the 21 eligible responders was 245 days. The encountered toxicities of rituximab were transient and most were of grade 1 or 2. Serum samples were collected for pharmacokinetic analysis in 12 patients. There was a steady increase in the peak and trough rituximab levels at all time points during the eight consecutive weekly treatments. It was concluded that rituximab monotherapy with eight consecutive weekly infusions showed significant anti-lymphoma activity in aggressive B-NHL with acceptable toxicities 18 ; . After the approval of rituximab in Japan in 2001, three kinds of new trials incorporating rituximab were initiated or are currently in preparation. One is a phase II III study of rituximab in combination with CHOP and biweekly CHOP for untreated advanced-stage, indolent B-NHL JCOG0203 ; . In the biweekly CHOP arm, an augmenting effect of antibodydependent cell-mediated cytotoxicity ADCC ; of rituximab is expected. Another study in preparation is a phase II study of HDC with AHSCT for untreated MCL, which expects to show an in vivo purging effect of rituximab. The third study in preparation is a phase II III study of rituximab in combination with CHOP for untreated diffuse large B-cell lymphoma, comparing weekly and tri-weekly administrations of rituximab. A schematic representation of the clinical trials of rituximab in Japan is shown in Fig. 3. Zenyaku Kogyo Co., Ltd, Tokyo, Japan, supported the clinical trials of rituximab.
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Side effects return to top side effects from dactinomycin are common and include: nausea and vomiting which may last up to 24 hours after treatment loss of appetite abdominal pain diarrhea difficulty swallowing thinned or brittle hair blistering skin or acne skin irritation sunburn-like ; or rash on areas previously exposed to radiation treatments tell your doctor if either of these symptoms is severe or lasts for several hours: fatigue mouth blistering if you experience any of the following symptoms, call your doctor immediately: unusual bruising or bleeding pain at the injection site persistent diarrhea or any change in normal bowel habits for more than 2 days fever chills cough sore throat dizziness shortness of breath yellowing of the skin or eyes if you experience a serious side effect, you or your doctor may send a report to the food and drug administration's fda ; medwatch adverse event reporting program online or by phone and danaparoid.
Background: Palliative care has been proposed to help meet the needs of patients who suffer progressive non-cancer conditions but there have been few evaluations of service development initiatives. We report here a novel protocol for the evaluation of a new palliative care service in this context. Methods Design: Using the MRC Framework for the Evaluation of Complex Interventions we modelled a new palliative care and neurology service for patients severely affected by Multiple Sclerosis MS ; . We conducted qualitative interviews with patients, families and staff, plus a literature review to model and pilot the service. Then we designed a delayed intervention randomised controlled trial to test its effectiveness as part of phase II of the MRC framework. Inclusion criteria for the trial were patients identified by referring clinicians as having unresolved symptoms or psychological concerns. Referrers were advised to use a score of greater than 8 on the Expanded Disability Scale was a benchmark. Consenting patients newly referred to the new service were randomised to either receive the palliative care service immediately fast-track ; or after a 12-week wait standard best practice ; . Face to face interviews were conducted at baseline before intervention ; , and at 46, 1012 before intervention for the standard-practice group ; , 16 18 and 2224 weeks with patients and their carers using standard questionnaires to assess symptoms, palliative care outcomes, function, service use and open comments. Ethics committee approval was granted separately for the qualitative phase and then for the trial. Discussion: We publish the protocol trial here, to allow methods to be reviewed in advance of publication of the results. The MRC Framework for the Evaluation of Complex Interventions was helpful in both the design of the service, methods for evaluation in convincing staff and the ethics committee to accept the trial. The research will provide valuable information on the effects of palliative care among non-cancer patients and a method to evaluate palliative care in this context.
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During the study period: 20.3% received atypicals only; 3.7%, conventionals only; and 3.6%, both atypicals and conventionals. Fewer than half 41.8% ; of treated residents received antipsychotic therapy in accordance with NH prescribing guidelines. One in four 23.4% ; patients had no appropriate indication, 17.2% had daily doses exceeding recommended levels, and 17.6% had both inappropriate indications and high dosing. Patients receiving antipsychotic therapy within guidelines were no more likely to achieve stability. The study used the data from the 20002001 Medicare Current Beneficiary Survey. High use does not necessarily indicate misuse, they said. However, "Our assessment detected high levels of apparent noncompliance with NH guidelines, " researchers said. "It is an open question whether the low compliance rates detected in this study represent justifiable differences of opinion between practicing physicians and the NH requirements for appropriate antipsychotic use, the researchers say. However, they noted, " this research demonstrates, as others have shown, that there are considerable gaps between the medications [that] clinical evidence recommends and the medications clinical practice delivers." Finally, the researchers said that their findings should be considered preliminary, and this issue deserves more investigation and dantrolene.
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Molecular Physiology and Pathophysiology of Electroneutral Cation-Chloride Cotransporters G. Gamba Physiol Rev, April 1, 2005; 85 ; : 423-493. [Abstract] [Full Text] [PDF] Calcium Absorption Across Epithelia J. G. J. Hoenderop, B. Nilius and R. J. M. Bindels Physiol Rev, January 1, 2005; 85 ; : 373-422. [Abstract] [Full Text] [PDF] Ion and diuretic specificity of chimeric proteins between apical Na + -K + -2Cl- and Na + -Clcotransporters C. Tovar-Palacio, N. A. Bobadilla, P. Cortes, C. Plata, P. de los Heros, N. Vazquez and G. Gamba J Physiol Renal Physiol, September 1, 2004; 287 ; : F570-F577. [Abstract] [Full Text] [PDF] Medline items on this article's topics can be found at : highwire anford lists artbytopic.dtl on the following topics: Biochemistry . Apical Membranes Medicine . Diuretics Physiology . Distal Convoluted Tubule Physiology . Anura Chemistry . Ion Binding Physiology . Rats Updated information and services including high-resolution figures, can be found at: : ajprenal.physiology cgi content full 279 1 F161 Additional material and information about AJP - Renal Physiology can be found at: : the-aps publications ajprenal and dapsone.
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At 1 and 3 yr, respectively, which was significantly lower than HCV patient survival at 97 and 93%. The clinical course of HCV infection in the transplant recipient has been the topic of study. Some authors suggested that HCV infection progresses more rapidly in the immunocompromised host, leading to cholestasis, fibrosing cholestatic hepatitis, and liver failure 5, 6 ; . Previous studies that were reviewed in the last transplantation NephSAP 7 ; indicated that the greater risk was cardiovascular disease mortality exacerbated by new-onset diabetes after transplantation. Perhaps the most important observation is that the mortality risk is higher when a transplant recipient is infected with HCV regardless of the event leading to death. Liver transplant recipients are at increased risk for chronic kidney disease discussed in CKD in Solid Organ Transplantation section ; . A GFR decline among such patients is customarily considered to be secondary to calcineurin inhibitor nephrotoxicity. Pillebout et al. 8 ; examined renal biopsies from 26 liver transplant recipients with chronic kidney disease and lowlevel proteinuria approximately 5 yr after transplantation. Impressive interstitial fibrosis, tubular atrophy, and glomerulosclerosis were found in all biopsies along with severe arteriosclerosis. Four underlying primary lesions were identified: 1 ; Calcineurin inhibitor nephrotoxicity, 2 ; diabetic nephropathy, 3 ; thrombotic microangiopathy, and 4 ; tubular changes attributed to a known previous administration of hydroxyethylstarch. Almost half of this cohort required renal replacement therapy. Conversely, McGuire et al. 9 ; reported that 25 of 30 liver transplant patients who underwent biopsy at the time of engraftment had an immune complex glomerulonephritis that was largely unsuspected clinically. Half of those affected had a typical membranoproliferative pattern of injury, 25% had IgA nephropathy, and 25% had mesangial glomerulonephritis. HCV infection has been associated with the latter primary nephritides in immunocompetent individuals. Taken together, such data indicate that HCV is associated with a variety of acute and chronic conditions that predispose individuals to kidney injury. Recurrence of a HCV-associated glomerulonephritis after transplantation has rarely been reported. Most recently, HCV also has been associated with myeloma 10 ; . In retrospective analysis of United Network for Organ Sharing data, the incidence of HCV infection was significantly higher in recipients who developed multiple myeloma after transplantation.
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Description Records the patient's preexisting medical conditions and or complications during the patient's hospital stay for the treatment of this cancer. Both are considered secondary diagnoses. Rationale Preexisting medical conditions and or complications may affect treatment decisions and influence patient outcomes. Information on comorbidities is used to risk adjust outcome statistics when evaluating patient survival and other outcomes. Complications may be related to the quality of care. Instructions for Coding If fewer than five comorbid conditions or complications are listed, then leave this data item blank. If only five comorbid conditions or complications are listed, then code the diagnoses listed and leave the remaining "Comorbidities and Complications" items blank. For further coding instructions, see Comorbidities and Complications #1 NAACCR Item #3110 and dalteparin.
FIG. 5. Effect of dactinomycin Act D ; or cycloheximide Cyclo ; on TCDD-induced cytochrome P-450 mRNA accumulation and nuclease sensitivity of the CYPIAJ gene in wild-type cells. A ; Cells were exposed to DMSO, TCDD, dactinomycin plus TCDD, dactinomycin alone, or cycloheximide plus TCDD. Nuclei were prepared, digested with BamHI B ; , AvaIl A ; , or RsaI R ; , and analyzed by using the B P probe. The membrane was exposed to XAR-5 film at -70C for 7 days in the presence of an intensification screen. B ; Wild-type cells were treated as described in the text. Total RNA was prepared as previously described 3 ; . A 10-, ug portion of total RNA was fractionated by gel electrophoresis 15 ; , transferred to Nytran, and hybridized with 32P-labeled CYPIAJ cDNA 21 ; . The membrane was exposed to XAR-5 film at -70C for 7 days in the presence of an intensification screen. The numbers indicate fragment size in kilobase pairs and daunorubicin.
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